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Discord Over DNA: Ideological Responses to Scientific Communication about Genes and Race1

Published online by Cambridge University Press:  14 August 2017

Alexandre Morin-Chassé
Affiliation:
Bureau of Institutional Research, University of Montreal
Elizabeth Suhay*
Affiliation:
Department of Government, School of Public Affairs, American University
Toby E. Jayaratne
Affiliation:
School of Public Health, University of Michigan
*
Address correspondence and reprint requests to: Elizabeth Suhay, Assistant Professor, Department of Government, School of Public Affairs, American University, Washington, D.C. E-mail: suhay@american.edu

Abstract

The American public's beliefs about the causes of social inequality vary greatly, with debates over the causes of racial inequality tending to be the most salient and divisive. Among whites in particular, liberals tend to see inequality as rooted in society's ills, whereas conservatives tend to see inequality as rooted in individuals’ shortcomings. Given this, many infer that white conservatives are more likely than white liberals to adopt the controversial view that racial inequality is “natural,” i.e., due to genetically inherited characteristics. We argue that genetic explanations for racial inequality, in and of themselves, offer little appeal to white conservatives. However, when white citizens are exposed to media messages that emphasize the egalitarian implications of genetic similarity between racial groups, those on the left and right engage in biased assimilation, resulting in a “nature” (conservative) versus “nurture” (liberal) divide. Data from two studies of white Americans—one representative survey and one experiment—support this theoretical framework.

Type
Research Article
Copyright
Copyright © The Race, Ethnicity, and Politics Section of the American Political Science Association 2017 

American conservatives and liberals disagree over how to respond to inequality. In recent decades, those on the left have been considerably more likely than those on the right to favor government policies that seek to remedy socioeconomic and racial inequality (Noel Reference Noel2013). This left-right difference over how to address inequality is accompanied by differences in the way those on the left and right explain inequality. Conservatives tend to make internal attributions, which blame individuals and, thus, justify opposition to government aid. Liberals tend to make external attributions, which—by blaming society—better justify governmental action (e.g., see Iyengar Reference Iyengar1991; Skitka et al. Reference Skitka, Mullen, Griffin, Hutchinson and Chamberlin2002).

Given this well-established pattern in the public at large, many infer that white conservatives are more likely than white liberals to adopt the controversial view that racial inequality is “natural,” i.e., due to inherited weaknesses, such as low intelligence.Footnote 2 This inference draws some support from the empirical literature. For example, at least two studies (Hunt Reference Hunt2007; Suhay and Jayaratne Reference Suhay and Jayaratne2013) find a positive relationship among whites only between conservatism (whether measured via policy support or self-identification) and the tendency to describe race-related characteristics as genetic in origin. However, the correlations are small in size, suggesting there may be heterogeneity at the individual level. Other research has called into question the association between a belief in the genetic determinism of racial differences and political ideology (e.g., Hochschild and Sen Reference Hochschild and Sen2015a).

In this article, we find room for both perspectives. We argue that the familiar “nature-nurture” debate that has at times pitted conservatives (arguing for nature) against liberals (arguing for nurture) does not dominate contemporary Americans’ thinking about socioeconomic and racial inequality. Today, genetic explanations for inequality offer less appeal to conservatives, and more appeal to liberals, than many researchers appreciate. However, we argue that the debate lies dormant and can easily reemerge. Following the literature on motivated reasoning and biased assimilation, we argue that white Americans on the left and right will diverge over whether racial inequality is “natural” when exposed to media messages that emphasize the egalitarian implications of genetic sameness and the inegalitarian implications of genetic difference. In other words, when offered a factual narrative that either justifies or undercuts important political goals, conservatives and liberals will gravitate toward the more “convenient” belief, in this case beliefs about what underlies racial inequality.

We test this intuition with two studies—one observational (Study 1) and one experimental (Study 2)—of white Americans’ explanations for black-white socioeconomic inequality. Both studies capitalize on a well-publicized scientific discovery: In 2000, researchers who had recently completed a first draft of the human genome announced that differences between traditional racial groupings were not reflected at the genomic level (NHGRI 2012). This information was widely disseminated and accompanied by a value frame that emphasized the egalitarian implications of this discovery.

Study 1 analyzes survey data collected shortly after the “race is not genetic” announcement. Study 2 analyzes experimental data in which treated participants were randomly assigned to read about the discovery. Both studies are consistent and support our theoretical framework: among white Americans not exposed to these media messages, conservatives and liberals do not differ in their tendency to view racial inequality as linked to biology; however, among those exposed to the messages, self-described liberals are considerably less likely than conservatives to say they believe racial inequality stems from biological differences between races. We discuss the implications in the section Discussion and Conclusion.

THEORETICAL FRAMEWORK

Internal versus External Explanations for Inequality

American conservatives and liberals tend to disagree about the causes of inequality. Generally speaking, conservatives tend to prefer explanations that are internal to individuals (also called “dispositional”). Liberals, on the other hand, tend to prefer external (or “situational”) explanations (Iyengar Reference Iyengar1991; Kluegel and Smith Reference Kluegel and Smith1986; Skitka et al. Reference Skitka, Mullen, Griffin, Hutchinson and Chamberlin2002). The accumulated evidence documenting this difference in attributional tendency between conservatives and liberals is so abundant that Skitka et al. have given it a name—the “ideo-attribution effect” (Morgan, Mullen and Skitka Reference Morgan, Mullen and Skitka2010; Skitka et al. Reference Skitka, Mullen, Griffin, Hutchinson and Chamberlin2002). This basic attributional difference between American conservatism and liberalism appears to be longstanding (Hofstadter Reference Hofstadter2006).

These differences are relevant to explanations for a number of different types of inequality, including (but not limited to) differences between blacks and whites in economic success, educational attainment, incarceration rates, etc. White conservatives are more likely than liberals to blame black Americans for poor life outcomes, and white liberals are more likely to blame discrimination or structural factors (Hunt Reference Hunt2007; Sniderman, Brody and Tetlock Reference Sniderman, Brody and Tetlock1991).

Why do conservatives and liberals disagree over what amounts to a factual question regarding the underlying causes of social inequality? Scholars argue that those on the left and right diverge over this question because different causes of inequality suggest different societal responses. Internal explanations for lack of success in life (e.g., lack of motivation) argue against government intervention, more in line with conservative preferences. External explanations (e.g., lack of access to jobs) justify government intervention, in line with liberal preferences. Government has a moral responsibility and pragmatic ability to solve problems caused by society, but this responsibility and ability diminish for problems caused by individuals (Brickman et al. Reference Brickman, Carulli Rabinowitz, Karuza, Coates, Cohn and Kidder1982; Iyengar Reference Iyengar1991; Suhay and Jayaratne Reference Suhay and Jayaratne2013).

While there is a logical relationship between many people's explanations for inequality and their political preferences, most scholars do not argue that different understandings of the causes of inequality (perhaps learned early in life) inform people's policy preferences. Rather, scholars tend to argue in favor of motivated cognition (Kunda Reference Kunda1990; Lodge and Taber Reference Lodge and Taber2013). That is, the attribution-politics link exists due to a kind of “backwards reasoning”: individuals seek to justify preexisting political preferences with congenial factual beliefs about the origins of inequality (Morgan, Mullen and Skitka Reference Morgan, Mullen and Skitka2010; Skitka et al. Reference Skitka, Mullen, Griffin, Hutchinson and Chamberlin2002).

Genetic Explanations for Inequality

Following recent scholarship on causal attributions (see especially Weiner, Osborne and Rudolph Reference Weiner, Osborne and Rudolph2011) and their associations with political attitudes (see, e.g., Suhay and Jayaratne Reference Suhay and Jayaratne2013), we emphasize the importance of unpacking the “internal” and “external” causal attribution categories to better understand their political implications. These two simple categories actually house a diverse range of explanations for inequality: the choices a person makes (internal); a person's biological predispositions (internal); the culture in which a person is situated (external); and social and structural discrimination (external).

This finer-grained examination makes clear that the political implications of the explanations within each category do not run cleanly in the same direction. The implications depend on who is blamed and who is best able to fix the problem. (See Brickman et al. Reference Brickman, Carulli Rabinowitz, Karuza, Coates, Cohn and Kidder1982; Iyengar Reference Iyengar1991; Weiner Reference Weiner1995.) While explanations that rest on choice align well with conservatism (the individual is blamed and able to remedy a lack of success) and explanations that point to discrimination align well with liberalism (society is to blame and able to remedy discrimination), genetic explanations are different.Footnote 3 Genetic explanations suggest no one is to blame—neither the individual nor society. And they suggest no one can easily solve the problem—because “genetic” characteristics are perceived to be immutable.Footnote 4 This suggests genetic explanations have both conservative and liberal implications. On the one hand, people are not to blame for their circumstances (liberal) (Dar-Nimrod and Heine Reference Dar-Nimrod and Heine2011; Weiner, Osborne and Rudolph Reference Weiner, Osborne and Rudolph2011). On the other hand, there is not much government can do to help (conservative) (Hofstadter Reference Hofstadter2006; Lewontin, Rose and Kamin Reference Lewontin, Rose and Kamin1984).

In other words, genetic explanations for inequality undermine conservatism to an extent. This is likely one reason why contemporary conservatives are far more likely to emphasize the role of personal responsibility (the choices a person makes) in one's success in life than inborn characteristics (see Brewer and Stonecash Reference Brewer and Stonecash2015). Further, the power of biology to “excuse” individuals for stigmatized characteristics helps to explain why those on the left are more likely than those on the right to make genetic attributions in some instances, including for homosexuality (Garretson and Suhay Reference Garretson and Suhay2016; Haider-Markel and Joslyn Reference Haider-Markel and Joslyn2008), obesity (Haider-Markel and Joslyn Reference Haider-Markel and Joslyn2017), and criminal behavior (Shen and Gromet Reference Shen and Gromet2015). Even with respect to racial inequality, there is potential for whites who believe genes are the cause to turn to paternalism, not antipathy—to “feel that blacks are simply incapable of improving their lot in life and, as a result, feel sympathy for them” (Brown et al. Reference Brown, Akiyama, White, Epstein Jayaratne and Anderson2009, 107).

Despite the above complications, relative conservatism and biological attributions for social inequality are in fact positively correlated among white Americans, if only modestly. Drawing on representative survey data from 2001, Suhay and Jayaratne (Reference Suhay and Jayaratne2013) find that white conservatives are about 10% more likely than white liberals to say that socioeconomic and racial inequality are due to genetic differences between groups. Analyzing General Social Survey data collected between 1985 and 2004, Hunt (Reference Hunt2007) also finds a relationship between conservatism and innate explanations for racial inequality among whites specifically.

Why these associations if the political implications of genetic explanations are so mixed? We argue that the on-average correlations between left-right political ideology and the belief that inequality is innate gloss over considerable heterogeneity in the American public.Footnote 5 We suspect that this heterogeneity stems in part from variation in exposure to media messages on this subject, messages that in recent years have tended to frame the nature-nurture debate in terms likely to divide left from right.

Scientific Communication about Genes and Race

Scholarly discussions about the causes of inequality have differed from discussions in the lay public in recent years. Academics have debated whether various types of inequality are due to “nature” and/or “nurture,” with little engagement with conservatism's core tenant of “personality responsibility” (see Segerstrale Reference Segerstrale2000). If blaming the individual or social group is not considered and the debate is restricted to blaming either nature or society, then the politics are simpler. If nature is an important causal force, then society (via government) has no obligation to solve inequality, nor would it be wise to try; if society is responsible, then government is morally obligated and able to provide a solution (Herrnstein and Murray Reference Herrnstein and Murray1994; Hofstadter Reference Hofstadter2006; Lewontin, Rose and Kamin Reference Lewontin, Rose and Kamin1984). Thus, among scholars, the argument that genes drive inequality has been understood as a conservative challenge to liberal policies.Footnote 6 The debate has been particularly heated surrounding the topic of race (Jacoby and Glauberman Reference Jacoby and Glauberman1995; Segerstrale Reference Segerstrale2000).

This debate took an important turn in 2000. In that year, upon “decoding” genomes of people from various racial groups, researchers discovered that genetic variation within each group far exceeded variation between the groups. Scientists concluded that race as commonly understood had no genetic basis. The scientists were not shy about publicizing these results and emphasizing what they perceived to be the egalitarian social and political implications (Bliss Reference Bliss2015). The findings were first announced at a White House press conference with President Bill Clinton and geneticists Dr. Craig Venter and Dr. Francis Collins. At the conference, Clinton said: “All of us are created equal, entitled to equal treatment under the law. After all, I believe one of the great truths to emerge from this triumphant expedition inside the human genome is that in genetic terms, all human beings, regardless of race, are more than 99.9 percent the same” (NHGRI 2012). The researchers echoed these egalitarian notions at the press conference and in follow-up interviews, stating explicitly that race was socially constructed and arguing that government policy should not be based on the faulty assumption that race differences are innate (e.g., McKie Reference McKie2001). The finding also generated an unusual amount of media coverage for a scientific topic, which we say more about in the empirical section. The dominant frame of the media coverage of this discovery followed the researchers’ lead: our skin color or racial lineage has nothing to do with our skills or talents (e.g., Angier Reference Angier2000). We are all born equal, after all.

Such value frames are common in science reporting and tend to be highly consequential with respect to how the public receives associated information (Conrad Reference Conrad, Asch, Beckwith, Conrad, Alper, Ard and Geller2002; Nisbet and Scheufele Reference Nisbet and Scheufele2009). However, such frames do not influence all members of the public equally. Value frames increase the likelihood that people will engage in “biased assimilation,” a type of motivated cognition whereby people filter new information according to their preexisting political beliefs and values (Kraft, Lodge and Taber Reference Kraft, Lodge and Taber2015; on biased assimilation generally, see, e.g., Kahan Reference Kahan2011; Lord, Ross and Lepper Reference Lord, Ross and Lepper1979). As a result, individuals are more likely to accept information they perceive to be in line with their political predispositions as compared with information that undermines them. Information that challenges predispositions can even lead to boomerang effects, as people devote mental resources to counterarguing the information and end up bolstering their priors (Hart and Nisbet Reference Hart and Nisbet2011). This framework suggests that white liberals—given their greater commitment to social equality—may have been more receptive to communications about the discovery that “race is not genetic” than white conservatives.

Our Studies

We focus, in two studies, on the beliefs and opinions of non-Hispanic whites.Footnote 7 We hypothesize that, among white Americans not exposed to media messages discussing scientists’ recent findings related to genes and race, there is no relationship between political ideology and genetic explanations for racial inequality (H1). Again, conservative rhetoric today argues in favor of personal responsibility—not genes—as a driver of unequal outcomes, and liberal rhetoric has increasingly made room for genetic influences (see above discussion). However, among those exposed to relevant media messages, we expect a left-right difference to emerge (H2): those on the left will be more likely than those on the right to find the egalitarian implications of genetic racial similarity attractive and to embrace scientists’ finding.

We carefully test these hypotheses by drawing on both observational and experimental data. In Study 1, we analyze survey data gathered several months after the “race is not genetic” discovery was announced. We investigate whether political ideology was correlated (in the expected direction) with genetic explanations for racial inequality only among those white Americans who reported paying attention to this topic. This first study possesses high external validity, demonstrating the relevance of our theoretical framework to real-world events. In Study 2, we follow-up with an experimental study in which American National Election Study (ANES) participants were randomly assigned to read one of the most prominent news stories covering the “race is not genetic” announcement. The controlled nature of this study complements the observational evidence by offering greater internal validity, i.e., a more rigorous test of the hypothesized causal relationship between media exposure and changing beliefs about race. Note that, to improve our ability to draw causal inferences regarding the role of the observed variables (media exposure and ideology in Study 1 and ideology in Study 2), we run many additional models with alternate specifications, including additional interaction terms. This allows us to be confident that the interactions we observe are not the result of a spurious association.

EMPIRICAL ANALYSES

Study 1

The initial announcement by genetic scientists that commonly used racial categories (African, Asian, Caucasian, etc.) did not map to corresponding genetic variation was made in June 2000. Despite the fanfare surrounding the announcement, it is possible that the discovery was not widely reported, as science topics tend to be underreported by media. In order to be sure that coverage of this event indeed reached a large portion of the public, we conducted an informal content analysis of newspaper coverage during the year following the announcement. See Appendix A for details. We find that significant related media coverage occurred in June and August/September of 2000 and again in February of 2001.

Study 1 analyzes cross-sectional survey data collected during the winter and spring of 2001. These data provide us with an unusual opportunity to test whether beliefs among Americans at the time were structured in the way we would expect if the assimilation of the scientific information available in the media was conditioned by political ideology.

Method

Sample

The study rests on a survey of Americans conducted in 2001 by researchers at the University of Michigan. We restrict our analyses to data from white participants (N = 600). The sample was obtained utilizing random-digit-dialing (RDD) methods, drawing from the continental U.S. (AAPOR Adjusted Response Rate 3 = 32%). Respondents were interviewed over the phone by professionally trained interviewers. Interviews averaged 40 min in length. Each respondent received $15 compensation (or $20 for refusal conversions). Within each household, adult respondents were randomly selected. Data are weighted for education and age (the sample is representative for gender).

Dependent Variable

The dependent variable focuses on participants’ explanations for stereotypical black-white differences that are commonly linked to socioeconomic inequality. Respondents were first asked to report whether they believe genetic differences play a role in explaining perceived racial differences (blacks versus whites) in drive to succeed. Respondents who answered “yes” were then asked to estimate how much of this difference is due to genetics. Responses to this pair of questions were recoded into five categories: “none” (0), “very little” (.25), “some” (.5), “a lot” (.75), or “just about all” (1). Respondents were then presented with three other pairs of questions, this time asking for their views about the role of genetics in explaining perceived racial differences in intelligence, mathematical abilities, and tendency toward violence. The resulting Genetic Attributions for Race Differences (GARD) scale represents the mean value of these four survey items (Cronbach's α = .85). In our sample, 565 respondents provided valid answers to all four questions. On average, GARD among our sample is relatively low (mean= 0.17; sd= 0.22). The exact wording of this and all other questions used in Study 1 is presented in Appendix C.

Independent Variables

Two independent variables are at the center of our attention. The first is a standard five-point political ideology scale with coding as follows: “very conservative” (0), “somewhat conservative” (.25), “middle of the road” (.5), “somewhat liberal” (.75), or “very liberal” (1). (We refer to political ideology—here as well as in Study 2—as liberalism to clearly indicate the direction of coding.) The second independent variable is a media exposure measure focused on the topic of genetics. Respondents were asked the following question: “Over the past few months, how often have you read or heard news stories about genetics in newspapers, magazines, or on TV?”Footnote 8 (Note that, in 2001, the internet had not yet emerged as an important news provider.) Response options were “often,” “sometimes,” “rarely” or “never.” Only 30 respondents answered “never.” We merge these respondents with those who chose “rarely” to form the low exposure group (n = 156). The respondents who answered “sometimes” are referred to as the moderate exposure group (n = 225), and those who said “often” are the high exposure group (n = 208). Our models use two dummy variables to distinguish the moderate and high exposure groups from the low exposure group. Note media exposure is not correlated with liberalism.Footnote 9

Controls

In this paper, we separate factual beliefs about the causes of perceived black-white differences from racial affect. In the contemporary United States, such factual beliefs are distinct both conceptually and empirically from a desire among whites for social distance from blacks as well as from racial resentment and symbolic racism (negative sentiment toward blacks justified by claims they disregard shared values) (Brown et al. Reference Brown, Akiyama, White, Epstein Jayaratne and Anderson2009). Even so, there is a modest relationship among these concepts (see Brown et al. Reference Brown, Akiyama, White, Epstein Jayaratne and Anderson2009; Jayaratne et al. Reference Jayaratne, Ybarra, Sheldon, Brown, Feldbaum, Pfeffer and Petty2006); thus, we control for racial prejudice in the models. Specifically, the full statistical models control for: social distance, which sums the answers to two questions asking respondents how bothered they would be if their son or daughter dated or married a black person; negative affect, which is measured by asking respondents how often they feel disgusted by blacks (see Banks and Valentino Reference Banks and Valentino2012); and contemporary prejudice, which is a seven-item additive scale (see Brown et al. Reference Brown, Akiyama, White, Epstein Jayaratne and Anderson2009) intended to gauge subtler forms of race prejudice (α = .74).

We also control for general knowledge about genetics, measured by summing the responses to four factual items. The full models also include standard demographic controls (religiosity, household annual income, education, age, and gender).

Note that all variables in the analyses have been re-coded to range from 0 to 1.

Results

The three models in Table 1 “predict” respondents’ location on the GARD scale using OLS regression. Note that here, and throughout the paper, p-values are one-tailed, given the directional nature of the hypotheses. We begin with a simple model without any interaction terms or control variables. Model 1 assesses the main effects of political ideology (liberalism) and media exposure. The coefficient on liberalism is negative and statistically significant (β = −.08, p <.01), reflecting previous research. The coefficients for media exposure are not significant. Thus, on average, media exposure did not reduce genetic explanations for race differences.

Table 1. Genetic attributions regressed on ideology, media exposure, and their interaction

Note: *p < .05;**p < .01;***p < .001, one-tailed tests. Unstandardized OLS regression coefficients. Standard errors are reported within parentheses.

Model 2 presents the main tests of our hypotheses (without controls). Among those with low media exposure, there is no clear relationship between ideology and genetic beliefs, as hypothesized (see the nonsignificant coefficient on liberalism, assessed at media exposure = 0). However, the coefficient for high exposure × liberalism is negative, substantial in size, and statistically significant (β = −.32, p < .001). This model also reveals a positive and significant coefficient for high exposure (β = .13, p < .01), which is assessed at liberalism = 0 (i.e., among very conservative participants). This indicates that strong conservatives in the high exposure group scored higher on the GARD scale than strong conservatives in the low exposure group. This suggests a boomerang effect occurred among the very conservative who were exposed to the “race is not genetic” news. In sum, exposure to news about genetics influenced strong liberals and conservatives in opposite ways, reducing GARD among the former and increasing it among the latter.Footnote 10 Overall, these patterns support our hypotheses.

Figure 1 presents the predicted values derived from Model 2. It is clear from the figure that the direction and the strength of the association between ideology (relative liberalism) and GARD depends on one's level of exposure to news about genetics. Among the high exposure group in particular, the width of the gap that separates very liberal (.01) and very conservative (.26) is strikingly large and statistically significant (p < .001).

Figure 1. This figure presents the interaction between political ideology and self-reported recent exposure to news about genetics. Shaded areas represent 95% confidence intervals. Predicted values were derived from Model 2, Table 1.

We ran additional models to verify the robustness of these general patterns. Model 3 adds numerous control variables to the core model.Footnote 11 While several of the coefficients on these variables are signed as expected and reach statistical significance (in particular, social distance and negative affect), the coefficient on high exposure × liberalism is not weakened by the inclusion of the controls.Footnote 12 In addition, the negative coefficient on the term moderate exposure × liberalism now reaches statistical significance (β = −.16, p < .05). Thus, the interactive pattern observed in Model 2 persists even when controlling for racial attitudes, knowledge about genetics, as well as various sociodemographic variables. Also, this model presents further evidence of a boomerang effect among strong conservatives. Indeed, we now observe that the coefficient for the term moderate exposure (i.e., the effect of moderate exposure, compared with low exposure, among the very conservative) is also positive and statistically significant. Finally, we would be remiss to avoid pointing out that this last model reveals a positive, significant coefficient for liberalism. Thus, among the low exposure group, net controls, strong liberals have a higher average GARD score than strong conservatives. We are hesitant to say much about this finding, given that it arises only in this model; however, it certainly is consistent with our contention that white conservatives are not uniformly more likely than white liberals to argue in favor of genetic explanations for black-white inequality.

Study 1 Discussion

Results from Study 1 reflect our expectations. Survey data gathered shortly after the discovery in 2000 that traditional racial categories had little to do with human DNA reveal that liberals were less likely than conservatives to argue in favor of genetic explanations for perceived racial differences only if they were exposed to science reporting on the subject. The data are strongly suggestive of ideologically biased assimilation, including a boomerang effect among strong conservatives.

Although our findings are consistent with the theoretical framework we have advanced, they do not allow us to rule out certain alternatives. First, the media exposure variable captures attention to news about genetic advances in general, not attention to the specific set of messages we discuss. Thus, the ideological patterns we see could be due to liberals and conservatives consuming different media content. Although the date of the study precedes the extreme era of media polarization in which we now find ourselves, conservatives in the sample were probably more likely than others to tune into Fox News or conservative talk radio and, with this in mind, the mechanism at work may be selective exposure to politically biased media (see Stroud Reference Stroud2011) rather than biased assimilation of similar media content.

A second, slightly different, explanation for our findings is that conservative and liberal news consumers reacted as they did because they were simply following partisan cues (e.g., Zaller Reference Zaller1992). While media frames of scientific findings are more likely to reference politically relevant values than political parties or specific politicians or actors (Nisbet and Scheufele Reference Nisbet and Scheufele2009), the original “race is not genetic” announcement admittedly occurred at a White House news conference during a Democratic administration. Our review of the news coverage that followed this event suggests that egalitarian values were emphasized far more than partisan cues; however, it remains possible that the presence of a Democratic cue in some news reports played an important role in diverging reactions among conservatives and liberals.

Third, some research indicates that political liberals are somewhat more trusting of scientists than conservatives (Blank and Shaw Reference Blank and Shaw2015; Gauchat Reference Gauchat2012). While recent research has called into question the assumption that only conservatives deny science that undercuts their values (Kahan Reference Kahan2013; Nisbet, Cooper and Garrett Reference Nisbet, Cooper and Garrett2015), it remains possible that we have simply observed a disparity in willingness to accept new scientific arguments.

Study 2, which draws on data from a randomized experiment embedded within the ANES, allows for a second test of our hypotheses while addressing these counterarguments.

Study 2

In this study, participants were randomly assigned to read or not to read a news article presenting the “race is not genetic” argument. Thus, the experimental design ensures that any interaction between political ideology and media exposure is due to politically biased responses to similar media content rather than selective exposure to different media content. This experiment also allows a more stringent test of whether political ideology—as opposed to partisanship—is driving the patterns observed in Study 1. Not only is the Study 2 stimulus free of partisan cues, but the survey component of the study contains a measure of partisanship (not available in Study 1), allowing us to control for this measure. Finally, the study also includes measures of trust in science and trust in the news media, allowing us to control for these possible confounds as well.

Method

Sample

Data were collected as a part of the American National Election Study (ANES 2014) Web Panel of 2008–9. Americans were sampled through stratified list-assisted RDD of landline telephone numbers. Participants were paid $10 per month to complete web questionnaires. The entire panel has 21 waves. Ten of the waves covered political topics; the remaining 11 were about a wider variety of issues and were partly funded by independent researchers. All data are publicly available on the ANES website.

Wave 16, collected in 2009, includes an experiment designed by Phelan, Link and Feldman (Reference Phelan, Link and Feldman2013). We take advantage of the public availability of these experimental data, using them for a different purpose. We examine whether exposure to a “race is not genetic” news article leads white liberals and conservatives to differentially alter their genetic attributions for black-white socioeconomic inequality. Our analyses bear on the 197 non-Hispanic white respondents assigned either to the control group (no treatment) or the news article condition relevant to our research question. Note that participants were balanced across the experimental groups on ideology and basic demographics. A description of the sample is in Table A4.

Dependent Variable

The dependent variable is a scale made up of averaged responses to three questions that are similar to the GARD items in Study 1. An advantageous difference between these and the Study 1 questions however is that the Study 2 questions specifically ask whether innate differences in certain behavioral characteristics are the cause of black Americans’ lower socioeconomic status. The first two questions were preceded by the statement that black Americans on average have “worse jobs, income, and housing” than whites; respondents were then asked the extent to which they believed this is due to (1) “less in-born ability to learn” and (2) “less in-born drive to succeed” among African Americans. A third question asked whether (3) African Americans’ greater involvement with the judicial system is “due to genetic differences between blacks and whites in their tendency toward violence.” These questions had four response options: “not at all” (0), “not much” (.33), “some” (.66), and “very much” (1). They were combined into an additive scale (α = .82). We refer to this as the Genetic Attributions for Black Inequality (GABI) scale. Like the GARD scale, on average, people scored somewhat low on the GABI scale (mean = .20; sd = .25). Note that the question wording for all Study 2 variables is presented in Appendix D.

Treatment

One of the most important news articles covering the “race is not genetic” thesis was by science journalist Nathalie Angier (Angier Reference Angier2000). The article was published in The New York Times on August 22, 2000 and drove subsequent print and broadcast media coverage. This particular article did an excellent job summarizing the new scientific consensus regarding the lack of association between racial groupings and genes as well as the egalitarian implications. The ANES experimental treatment is an adaptation of this article.

The treatment includes statements, which are either identical or nearly identical to the original news article. According to the scientist cited in the treatment article, humans share 99.9% of their DNA and there exists greater genetic variation within racial groups than between them. Physical differences such as skin color are due to a small number of genes, which are sensitive to fast changing environmental pressures. In contrast, any genetic influences on complex social traits like intelligence involve thousands of genes interacting in complex ways. The scientist concludes: “This is why the differences that we see in skin color do not translate into widespread biological differences that are unique to groups and why (…) the standard labels used to distinguish people by ‘race’ have little or no biological meaning.” Note that the treatment article (see Appendix D) contains no mention of the White House news conference or of political elites’ positions on either side of the political aisle.

In our analyses, a treatment dummy variable is coded 1 for treatment group, and 0 for the control group. Those in the control group received no article.

Independent Variable

Ideology, measured in a previous panel wave, is a seven-point scale coded “very conservative” (0), “somewhat conservative” (.166), “leaning conservative” (.333), “Neither conservative nor liberal” (.5), “leaning liberal” (.666), “somewhat liberal” (.833), and “very liberal” (1).

Control Variables

Again, some of our models control for possible confounds to test the robustness of our findings. While controls are less important in a randomly assigned experiment than a survey, the addition of control variables here helps in three ways: (1) while the treatment is randomly assigned, ideology is not randomly assigned; thus, covariates with ideology are particularly suspect as confounds; (2) controls used in experimental analyses generally improve the efficiency of regression estimations (Bowers Reference Bowers, Kuklinski, Druckman, Green and Lupia2011); (3) we can test whether additional interaction terms alter the results from the original model.

We measured positive affect toward blacks by taking the mean of responses to two questions asking how often respondents feel sympathy and admiration for black people (Pearson's r = .27). Contemporary prejudice was measured with six questions (α = .761) that measure the same type of subtle prejudice as in Study 1 (see Neville et al. Reference Neville, Lilly, Duran, Lee and Browne2000). It is possible that participants’ reactions to news about genetics depend on their attitudes toward the source of the message; thus, we control for participants’ trust in scientists and trust in news.Footnote 13 We also control for the same socioeconomic variables (religiosity, education, income, age, sex) as in Study 1. Finally, we examine the influence of party identification (standard seven-point scale).

Again, all variables have been re-coded to range from 0 to 1.

Results

Table 2 presents three OLS regression models. Again, the statistical thresholds marked in the tables represent one-tailed tests. In Model 1 (direct effects), liberalism is negatively associated with the GABI scale (β = −.13, p < .05), but the treatment has no on average effect. In Model 2 (interactive effect), the coefficient on liberalism (reflecting participants in the control group) is equivalent to zero; however, the treatment × liberalism interaction term is negative, significant, and fairly large (β = −.21, p < .05). In other words, the on-average negative effect for liberalism in Model 1 is completely driven by the treatment group. These findings—the direct and indirect effects—mirror those from Study 1.

Table 2. Effect of treatment & ideology on genetic attributions for black inequality

Note: *p < .05;**p < .01;***p < .001, one-tailed tests. Unstandardized OLS regression coefficients. Standard errors are reported within parentheses.

Figure 2 presents the predicted values derived from Model 2 (Table 2). Among the control group, the slope for ideology (relative liberalism) is flat; in contrast, for those in the treatment group, the slope is negative. Among those exposed to the race is not genetic experimental condition, the average score on the GABI scale for very conservative participants equals .28. The equivalent value for very liberal participants is .08. The difference between these two values is statistically significant (p <.01).

Figure 2. This figure presents the interaction between political ideology and exposure to the treatment news article. Shaded areas represent 95% confidence intervals. Predicted values were derived from Table 2, Model 2.

We again ran additional models to test the robustness of these results. We focus on Model 3, which adds control variables to the interactive model. Model 3 confirms that the key interactive relationship is robust to the addition of controls.Footnote 14 Indeed, the absolute value of the coefficient on treatment × ideology is larger in size in this model (β = −.32, p < .01). Further, the coefficient on the treatment variable alone (assessed at liberalism = 0) suggests another boomerang effect among the very conservative (β = .11, p < 05).Footnote 15

We provide additional tests of the robustness of the treatment × liberalism interactive effect by introducing additional interaction terms into the model. The models in Table A6 confirm that the effect of our key interaction term cannot be reduced to moderation by contemporary prejudice, positive affect towards blacks, trust in scientists, trust in news, education or age. In Table A7, we add an interaction between party identification and treatment exposure. Party identification has no significant effect on GABI in the control or treatment group, and the coefficients on our key ideology interactive term actually increases in size. In sum, the negative treatment × liberalism interactive effect is quite robust.

Study 2 Discussion

The aim of this study was to test our initial hypotheses in a more controlled environment. The results again support our two hypotheses. Without exposure to science reporting on race, relative conservatism among white Americans was not associated with genetic explanations for racial inequality; however, the expected ideological pattern emerged among those exposed to news on the “race is not genetic” finding.

Study 2 achieves this objective by manipulating the information presented to participants rather than relying on self-reported media exposure. Thus, the experiment suggests that the effects observed in Study 1 were not solely caused by ideologically driven selective exposure. The follow-up analyses also show that the observed patterns (A) are not driven by different levels of trust in science or the media, and (B) are driven by biased assimilation of information based on political ideology, not party.

DISCUSSION AND CONCLUSION

We argue that much heterogeneity underlies the correlation among U.S. whites between conservatism and genetic explanations for perceived racial differences and inequality. This heterogeneity is structured in a way that suggests an explanation for why conservatives and liberals differ on this question on average. In short, when exposed to media messages that associate genetic sameness with racial egalitarianism, white conservatives and liberals diverge over the import of “nature”: conservatives are more likely to embrace the idea that genes drive racial inequality and associated stereotypes, and liberals to reject it. Findings from two studies of random samples of U.S. whites—one observational and one experimental—support this framework and are robust to a variety of counterarguments, including selective exposure to media, differential trust in science or the media, and party cues.Footnote 16

We also demonstrate that ideology conditioned acceptance of the “race is not genetic” message regardless of whether or not we controlled for racial attitudes, including a desire for social distance from blacks, affect toward blacks, and contemporary racial prejudice. Thus, this article points to the utility of “unhooking” factual beliefs about the origins of racial differences (real and perceived) from measures of racial attitudes. Throughout our analyses, racial prejudice is indeed associated with genetic explanations, in line with previous scholarship; however, relevant media messages in conjunction with ideology also have a strong and consistent effect on genetic explanations that is independent of racial prejudice. In short, we propose that value differences between liberals and conservatives can lead to biased uptake of information about whether human differences are natural or socially constructed.

What are the implications? On the one hand, our article challenges some assumptions about political conservatives. Many scholars assume that white conservatives’ tendency to commit what some psychologists call the “fundamental attribution error” (blaming individuals for perceived deficiencies; see Ross Reference Ross and Berkowitz1977) also means that they will commit the “ultimate attribution error” (blaming outgroups’ genes for perceived deficiencies; see Byrd and Ray Reference Byrd and Ray2015; Pettigrew Reference Pettigrew1979). Our evidence suggests that, all else equal, this is not the case. Genes may be internal to the individual, but they also are understood by many as absolving individuals of blame for associated characteristics and behaviors. On average, this decreases their appeal to conservatives, and increases their appeal to liberals.

This said—in line with growing evidence for the biased assimilation of factual information with political relevance—white conservatives and liberals do diverge if exposed to media messages that mute this complicating aspect of genetic explanations, instead emphasizing their racially inegalitarian implications. The origin of this factual polarization is likely white conservatives’ and liberals’ sharp disagreement over the importance of using government action to address social inequality, perhaps racial inequality in particular. Given our argument that these patterns are not necessarily specific to beliefs about racial differences, similar patterns ought to emerge in response to comparable scientific messages about the origins of other types of inequality, such as differences between wealthy and poor or between women and men. But we cannot test this intuition with our data, leaving it for future research.Footnote 17

Even if these motivated reasoning patterns stem from a support for, or wariness of, egalitarian policy generally, there are obvious and important parallels to the way in which racial prejudice has evolved in the United States since the mid-twentieth century. Scholars describe a decline in “old-fashioned racism” (including its biological component) after World War II but a simultaneous increase in the notion that black Americans are morally inferior, choosing laziness over hard work (Kinder and Sanders Reference Kinder and Sanders1996). In the same way that we document attributional beliefs “moving around” in defense of political ideology, others argue they have moved around conveniently to defend racial hierarchy (e.g., Bonilla-Silva Reference Bonilla-Silva2014). Maybe this is a distinction without much of a difference, as even conservatism drained of the racial animus with which it is intertwined in the real world still supports socioeconomic hierarchy (Jost et al. Reference Jost, Glaser, Kruglanski and Sulloway2003). But, to us, the distinction is worth making: whatever its complicated origins, American conservatism has long emphasized personal responsibility, an idea that is not logically well-suited to genetic explanations for behavior, which undercut the whole notion of individual agency. To bring conservatism to genes requires a little work. We do not rule out the possibility that conservatism itself may change in the process.

With this in mind, it is important to acknowledge the contextual nature of our studies: we focus on reactions to a specific scientific message that was widely distributed in the wake of the decoding of the human genome around the turn of the millenium, and data from our studies were collected in 2001 and 2009. We encourage other researchers to examine more recent trends. For example, the ethno-nationalist movement sometimes referred to as the “alt-right” has become a powerful force on social media, emboldened in part by the explicit prejudice and xenophobia of Donald Trump's presidential campaign. There is no question that many members of the alt-right endorse the idea that black Americans (as well as many other groups) are genetically inferior to white Christians (Matthews Reference Matthews2016). As they mix conservative political rhetoric with these factual claims of natural inferiority, it is likely that such ideas will resonate with some political allies on the right. With this in mind, we would not be surprised if the association between conservatism (and, perhaps, Republicanism) and genetic explanations for perceived racial difference and inequality has grown in the last couple of years, particularly among those attentive to new media.

In sum, we argue that individuals’ political viewpoints drive their factual beliefs about the world much more than most care to admit, and this includes explanations for inequality. At the same time, it is important to recognize that ordinary citizens’ belief systems are not necessarily organized in a manner similar to political or scientific elites. However, citizens who are paying attention will tend to follow the lead of elites in terms of “what goes with what” (Converse Reference Converse and Apter1964; Zaller Reference Zaller1992). From a normative perspective, this should serve as a cautionary tale to elites who communicate scientific facts to the public. Even those who have the best of intentions when they point out egalitarian implications of new knowledge may contribute to political polarization.

ACKNOWLEDGEMENTS

We are grateful to Paul Appelbaum, Kevin Arceneaux, Matthew Green, Jen Lawless, Jan Leighley, Matt Levendusky, Tali Mendelberg, Hans Noel, and Richard Skinner for helpful comments and advice. Part of this work was supported by the National Human Genome Research Institute (HG01881 to T.E.J.).

Appendix A: Analysis of Media Coverage

To gauge the extent of media coverage of the Human Genome Project / Celera Genetics Corporation finding regarding race, we conducted a search of four media databases (Factiva, Infotrack Newsstand, Newsbank, and Google News) for newspaper articles using relevant keyword combinations (e.g., genetics, race, 99.9%, Craig Venter).Footnote 18 We focus on the year following the announcement (from June 25 2000 to June 30 2001). Each article was extracted and read by one of the authors to ensure that it addressed the “race is not genetic” finding; those that did not were excluded from the results.

During this time period, Americans were exposed to three waves of news related to this announcement. The first wave occurred in the days that followed the White House press conference. Reports in most major newspapers, such as The New York Times, The Boston Globe, and Chicago Sun-Times mentioned the finding about race. Many regional or local newspapers also covered the conference and the issue of genetics and race. The second wave of news occurred a few weeks later. On August 22, The New York Times published an influential news article entitled “Do Races Differ? Not Really, Genes Show.” Reporter Nathalie Angier interviewed several scientists and argued that “most of them are convinced that the standard labels used to distinguish people by ‘race’ have little or no biological meaning.” In the days that followed, at least 14 regional or local newspapers (e.g., The Denver Post (CO), Tulsa World (OK), St Louis Post-Dispatch (MO), The Star-Ledger (NJ)) quoted different parts of Angier's news article. A third wave began in February 2001 after the publication of two new research articles (in Science and Nature) supporting the “race is not genetic” finding and the holding of another public conference by the Human Genome Consortium. This finding received coverage in The Washington Post, USA Today, and The New York Times, among others.

Table A1. Regression models by subgroups of media exposure (study 1)

Table A2. Controlling for potential confounds of the political ideology term (study 1)

Table A3. Controlling for potential confounds of the media exposure term (study 1)

Table A4. Sample description (study 2)

Table A5. Regression models by experimental subgroups (study 2)

Table A6. Controlling for other interactions (study 2)

Table A7. Ideology versus partisan identification (study 2)

Appendix B: Additional Tables

Appendix C: Study 1 Measures

Questions Used to Generate the GARD Scale

Some people think [A: whites TEND to differ from Blacks] [B: blacks TEND to differ from whites] in their DRIVE TO SUCCEED. Do YOU think their genes have ANYTHING to do with THIS difference?

Yes, No, No difference VOL

(if yes) How much of THIS difference between whites and Blacks do YOU think is due to their genes?

Very Little, Some, A lot, Just about all

Some people think [A: whites TEND to differ from Blacks] [B: blacks TEND to differ from whites] in how good they are in MATH. Do YOU think their genes have ANYTHING to do with THIS difference?

Same answers as above

(if yes) How much of THIS difference do YOU think is due to their genes?

Same answers as above

Some people think [A: whites differ from Blacks] [B: blacks differ from whites] in their TENDENCY TO ACT VIOLENTLY. Do YOU think their genes have ANYTHING to do with THIS difference?

Same answers as above

(if yes) How much of THIS difference do YOU think is due to their genes?

Same answers as above

Some people think [A: whites TEND to differ from Blacks] [B: blacks TEND to differ from whites] in INTELLIGENCE. Do YOU think their genes have ANYTHING to do with THIS difference?

Same answers as above

(if yes) How much of THIS difference do YOU think is due to their genes?

Same answers as above

Political Ideology

Compared with other people, do YOU generally think of yourself POLITICALLY, as very liberal, somewhat liberal, middle-of-the-road, somewhat conservative or very conservative?

Very liberal, Somewhat liberal, Middle of the road, Somewhat conservative, Very conservative

Media Exposure

Over the past few months, how often have you read or heard news stories about genetics in newspapers, magazines or on TV? Would you say…

Often, Sometimes, Rarely, Never

Social Distance

First, how bothered would you be if your son or daughter DATED a Black person?

Not bothered at alltovery bothered

How bothered would you be if your son or daughter MARRIED a Black person?

Same as above

Negative Affect

Next, people have different feelings toward some groups in our society. I'm going to read you a list of groups and I'd like you to tell me how often you have certain feelings toward them. We're not asking your feelings about ANY SPECIFIC person in the group, but how you feel about the GROUP OF PEOPLE, IN GENERAL …

How about Blacks?

Often, Sometimes, Rarely, Never

Questions Used to Generate the Contemporary Prejudice scale

Again, please tell me if you STRONGLY AGREE, SOMEWHAT AGREE, SOMEWHAT DISAGREE or STRONGLY DISAGREE with the following statements.

  • Society has reached the point where Blacks and Whites have equal opportunities for success. (reversed)

  • Discrimination against Blacks is a serious problem in the United States.

  • Whites have an unfair advantage in our society.

  • Many groups of Americans overcame discrimination and made it on their own. Blacks should do the same. (reversed)

  • If Blacks don't do well in life, they have only themselves to blame. (reversed)

  • In this society, Whites have many more opportunities to get ahead than Blacks.

Knowledge about Genetics

When they're born, identical twins have exactly the same genes. (reversed)

Strongly agree, Somewhat agree, Neither agree/disagree VOL, Somewhat disagree, Strongly disagree

Single genes directly control specific human behaviors.

Same answers as above

On average, a person has half their genes in common with their siblings. (reversed)

Same answers as above

There are different types of genes in different parts of the body.

Same answers as above

Religiosity

Generally do you think of yourself as very religious, somewhat religious, not very religious or not religious at all?

Very religious, Somewhat religious, Not very religious, Not religious at all

Education

Derived from: What is the highest grade of school or year of college you've completed? What is the highest degree that you have earned?

Less than 12th grade, Graduated high school or equivalent, Some college, Associate's degree, Bachelor's degree, Master's degree, Professional or doctoral degree

Income

Now, including yourself and all family members in your household, what was your total family income for 2000 before taxes? For this question we just need a range. Stop me when I read the right category…

Less than $5,000, $5–10,000 [9,999], $10–20,000 [19,999], $20–30,000 [29,999], $30–40,000 [39,999], $40–50,000 [49,999], $50–60,000 [59,999], $60–70,000 [69,999], $70–80,000 [79,999], $80–100,000 [99,999], $100,000 or more

Age

Derived from: In what year were you born?

Appendix D: Study 2 Treatment and Measures

Treatment Article

Is Race Real? Genes Say ‘No’

Most people would agree it is easy to tell at a glance if a person is Caucasian, African or Asian.

But a recent study suggests that it is not so easy to make these distinctions when one probes beneath surface characteristics and looks for DNA markers of “race.”

Results of the study were published yesterday in the journal Nature Genetics. The study was conducted by Dr. Bruce Firman and other geneticists at Columbia University.

Analyzing the genes of people from around the world, the researchers found that the people in the sample were about 99.9 percent the same at the DNA level. “That means that the percentage of genes that vary among humans is around .01 percent, or one in ten thousand. This is a tiny fraction of our genetic make-up as humans,” noted Dr. Firman.

The researchers also found that there is more genetic variation within each racial or ethnic group than there is between the average genomes of different racial or ethnic groups.

Why the discrepancy between the ease of distinguishing “racial” groups visually and the difficulty of distinguishing them at a genetic level?

Traits like skin and eye color, or nose width are controlled by a small number of genes. Thus, these traits have been able to change quickly in response to extreme environmental pressures during the short course of human history.

But the genes that control our external appearance are only a small fraction of all the genes that make up the human genome.

Traits like intelligence, artistic talent, and social skills are likely to be shaped by thousands, if not tens of thousands of genes, all working together in complex ways. For this reason, these traits cannot respond quickly to different environmental pressures in different parts of the world.

This is why the differences that we see in skin color do not translate into widespread biological differences that are unique to groups and why Dr. Firman says “the standard labels used to distinguish people by ‘race’ have little or no biological meaning.”

Measures

Questions Used to Generate the GABI Scale

On average, black people in the United States have worse jobs, income, and housing than white people. There are many possible explanations for these differences. How much do you think these differences are …

because most African Americans have less in-born ability to learn?

Very much, Some, Not much, Not at all

because most African Americans have less in-born drive to succeed?

Same answers as above

African Americans are much more likely to be arrested, jailed and imprisoned in the United States than are whites. There are many possible explanations for these differences. How much do you think these differences are …

due to genetic differences between blacks and whites in their tendency toward violence?

Same answers as above

Political Ideology

When it comes to politics, would you describe yourself as liberal, conservative, or neither liberal nor conservative?

Liberal, Conservative, Neither liberal nor conservative

IF LIBERAL:

Would you call yourself very liberal or somewhat liberal?

Very liberal, Somewhat liberal

IF CONSERVATIVE:

Would you call yourself very conservative or somewhat conservative?

Very conservative, Somewhat conservative

IF NEITHER LIBERAL NOR CONSERVATIVE:

Do you think of yourself as closer to liberals, or conservatives, or neither of these?

Closer to liberals, Closer to conservatives, Neither of these

Party Identification

Generally speaking, do you usually think of yourself as a Republican, a Democrat, an independent, or what? (rotate 1 and 2)

Republican, Democrat, Independent, Something else

Would you call yourself a strong [Democrat/Republican] or a not very strong [Democrat/Republican]?

Strong [Democrat/Republican], Not very strong [Democrat/Republican]

Do you think of yourself as closer to the Republican Party or to the Democratic Party?

Closer to the Republican Party, Closer to the Democratic Party, Neither

Contemporary Prejudice

Racial problems in the United States are rare, isolated situations.

Strongly agree, Somewhat agree, Somewhat disagree, Strongly disagree

Talking about racial issues causes unnecessary tension.

Same answers as above

Racism is a major problem in the United States.

Same answers as above

It is important for public schools to teach about the history and contributions of racial and ethnic minorities.

Same answers as above

It is important for political leaders to talk about racism to help work through or solve society's problems.

Same answers as above

Racism may have been a problem in the past, it is not an important problem today.

Same answers as above

Positive Affect

How often have you felt sympathy for blacks?

Always, most of the time, about half the time, once in a while, or never?

How often have you felt admiration for blacks?

Always, most of the time, about half the time, once in a while, or never?

Trust

Please tell us how much confidence you have in the following groups and institutions in this country. In general, would you say you have no confidence at all, a little confidence, a moderate amount of confidence, a lot of confidence, or a great deal of confidence in:

  • University scientists

  • Newspapers

Religiosity

How often do you attend religious services, watch religious programs on television or listen to them on the radio, or visit religious websites?

Daily, Weekly, At least once a month, Now and then, Never

Education

Derived variable from the ANES 2008–2009 Panel Dataset

No high school diploma, High school diploma, Some college, no bachelor's degree, Bachelor's degree, Graduate degree

Income

Derived variable from the ANES 2008–2009 Panel Dataset

Less than $5,000, $5,000 to $7499, $7,500 to $9,999, $10,000 to $12,499, $12,500 to $14,999, $15,000 to $19,999, $20,000 to $24,999, $25,000 to $29,999, $30,000 to $34,999, $35,000 to $39,999, $40,000 to $49,999, $50,000 to $59,999, $60,000 to $74,999, $75,000 to $84,999, $85,000 to $99,999, $100,000 to $124,999, $125,000 to $149,999, $150,000 to $174,999, $175,000 or more

Age and Gender

Derived variables from the ANES 2008–2009 Panel Dataset

Footnotes

1.

Authors’ Note: The first two authors contributed equally to this article.

2. Note that many scholars would label this set of beliefs “biological racism.” In using more plain language, we do not deny the empirical association between these beliefs and racial prejudice. Particularly prior to World War II, a belief in the inherent physical inferiority of black Americans served to justify racial segregation and restricted political rights. We say more about this topic when we describe our studies as well as in the final discussion section.

3. Cultural explanations are also complicated politically. While cultural influences are “outside” the individual, they simply shift blame to a cultural group. Although, some people who argue in favor of cultural explanations of inequality ultimately “blame” historic discrimination.

4. See Dar-Nimrod and Heine (Reference Dar-Nimrod and Heine2011) and Morin-Chassé (Reference Morin-Chassé2014) on ways in which the lay public misunderstands the influence of genetics on human behavior. One common misperception is that genes have a direct and unavoidable influence on behavior. In reality, genetic influences are highly complex and conditional on gene–gene and gene–environment interactions.

5. The fact that low correlations often disguise heterogeneous patterns in the population is a problem that deserves wider recognition (for a helpful recent discussion, see Feldman Reference Feldman, Sears, Huddy and Levy2013).

6. It is worth noting that academics, particularly in more left-leaning disciplines, appear to be more cognizant than the general public of the historical linkages between genetic explanations and discrimination, including eugenic policy (see Hochschild and Sen Reference Hochschild and Sen2015b). This is yet an additional reason why scholars in particular have interpreted genetic explanations as anti-egalitarian.

7. We do not mean to imply that nonwhites’ beliefs about this issue are uninteresting. Rather, previous research has found that the modest on-average relationship between conservatism and genetic explanations for racial difference occurs only among white subsamples. There are at least two reasons why this may be the case. First, political ideology—in particular, the meaning of “conservatism”—among black Americans differs in significant ways from white Americans (Dawson Reference Dawson2003). White conservatism is distinct in part for its opposition to government efforts to address social (including racial) inequality. Second, genetic explanations for black-white inequality are both more personal, and likely offensive, to black Americans. Separate analyses of an oversample of black Americans from Study 1 confirms that the observed patterns do not occur among black Americans. Relevant analyses are available upon request.

8. We are aware of criticisms of such self-assessed media exposure measures. However, such measures, while flawed, provide useful information that is more reliable than many argue (see Dilliplane, Goldman and Mutz Reference Dilliplane, Goldman and Mutz2013; Goldman, Mutz and Dilliplane Reference Goldman, Mutz and Dilliplane2013). Alternative measures of exposure, such as knowledge, are inappropriate for a study on biased assimilation of information (Kahan Reference Kahan2015). Finally, note that Study 2—which randomly assigns media content to participants—confirms the Study 1 findings.

9. An ANOVA revealed no mean difference in ideology across media groups ( $ prob \gt F{\rm = }.94$ ).

10. Recall that the dependent variable is an additive scale aggregating the answers to four pairs of survey questions. Additional OLS regression analyses confirm that the patterns observed in Table 1 are also observed when we run the models for each of the scale items separately.

11. A more stringent test would also control for competing interactive relationships. Perhaps the media exposure × ideology interaction actually reflects media exposure × racial prejudice. Or, alternatively, perhaps the coefficient for ideology varies by education or knowledge about genetics, rather than media attention, suggesting a sophistication effect (Lodge and Taber Reference Lodge and Taber2013) not a media exposure effect. We tested these counterarguments by adding additional interaction terms to the models. The results, presented in Tables A2 and A3 in Appendix B, offer no support for these counterarguments.

12. For additional regression models where observations are restricted to each media exposure group separately, see Table A1.

13. Experimental studies conditioning results on attitudinal measures should use measures assessed pre-treatment to avoid bias. The questions making up the prejudice scales were assessed prior to the experimental manipulation. The trust questions were asked post-treatment; however, they were assessed a full 2 months later. As one might expect, the experimental manipulation did not impact responses to these questions (trust in scientists, p = .42; trust in news, p = .50), making us confident that the inclusion of these post-treatment variables does not bias our estimates.

14. In this model, coefficients on the control variables are signed as previous research and intuition would have us expect (for example, see contemporary prejudice and trust in scientists) but do not reach statistical significance; this is attributable in part to the relatively small sample size (and, thus, low power) of Study 2.

15. As was the case in Study 1, running the models presented in Table 2 on each individual item of the GABI scale separately revealed that the interactive pattern is consistent across items.

16. One counterargument is that our findings reveal political correctness by liberals (i.e., they are less honest after reading messages that genes do not cause racial difference). Previous researchers (Suhay and Jayaratne Reference Suhay and Jayaratne2013) examined the Study 1 dataset for evidence of political correctness—including different patterns of response when the interviewer was believed to be black—and found none. Further, research shows social desirability concerns are low in online studies (Kreuter, Presser and Tourangeau Reference Kreuter, Presser and Tourangeau2008), such as Study 2.

17. We hope future researchers will test this framework to better understand its reach as well as its underlying mechanisms. A promising direction for research may be assessing the specific cognitions and emotions that are aroused in response to the type of information we examine in this study.

18. We do not claim that all Americans read newspapers or that the only source for such information was a newspaper. Rather, at least prior to the current “new media” age, newspaper coverage was roughly indicative of the media environment of the time.

Note: *p < .05;**p < .01;***p < .001, one-tailed tests. Unstandardized OLS regression coefficients. Standard errors in parentheses.

Note: *p < .05;**p < .01;***p < .001, one-tailed tests. Unstandardized OLS regression Standard errors are reported within parentheses.

aThe other control variables included are knowledge, religiosity, education, income, age, and gender.

Note: *p < .05;**p < .01;***p < .001, one-tailed tests. Unstandardized OLS regression Standard errors are reported within parentheses.

aThe other control variables included are social distance, negative affect, contemporary prejudice, income and gender.

Note: *p < .05;**p < .01;***p < .001, one-tailed tests. Unstandardized OLS regression coefficients. Standard errors are reported within parentheses.

Note: *p < .05;**p < .01;***p < .001, one-tailed tests. Unstandardized OLS regression Standard errors are reported within parentheses.

aThe other control variables included are religiosity, income, and gender.

Note: *p < .05;**p < .01;***p < .001, one-tailed tests. Unstandardized OLS regression Standard errors are reported within parentheses.

aThe other control variables included are contemporary prejudice, positive affect, trust in science, trust in news, religiosity, education, income, age, and gender.

References

REFERENCES

ANES (American National Election Study). 2014. 2008–2009 Panel Study [dataset]. Stanford University and the University of Michigan.Google Scholar
Angier, Natalie. 2000. “Do Races Differ? Not Really, Genes Show,” New York Times, 22:F1.Google Scholar
Banks, Antoine J., and Valentino, Nicholas A.. 2012. “Emotional Substrates of White Racial Attitudes.” American Journal of Political Science 56 (2): 286–97.Google Scholar
Blank, Joshua M., and Shaw, Daron. 2015. “Does Partisanship Shape Attitudes Towards Science and Public Policy? The Case for Ideology and Religion.” ANNALS of the American Academy of Political and Social Science 658 (1): 1835.CrossRefGoogle Scholar
Bliss, Catherine. 2015. “Science and Struggle Emerging Forms of Race and Activism in the Genomic Era.” ANNALS of the American Academy of Political and Social Science 661 (1): 86108.Google Scholar
Bonilla-Silva, Eduardo. 2014. Racism without Racists: Color-blind Racism and the Persistence of Racial Inequality in America, Fourth Edition. New York: Rowman & Littlefield.Google Scholar
Bowers, Jake. 2011. “Making Effects Manifest in Randomized Experiments.” In Cambridge Handbook of Experimental Political Science, eds. Kuklinski, James H., Druckman, James N., Green, Donald P., and Lupia, Arthur. New York: Cambridge. pp. 459–80.Google Scholar
Brewer, Mark D., and Stonecash, Jeffrey M.. 2015. Polarization and the Politics of Personal Responsibility. New York: Oxford University Press.Google Scholar
Brickman, Philip, Carulli Rabinowitz, Vita, Karuza, Jurgis Jr., Coates, Dan, Cohn, Ellen, and Kidder, Louise. 1982. “Models of Helping and Coping.” American Psychologist 37 (4): 368–84.Google Scholar
Brown, Tony N., Akiyama, Mark K., White, Ismail K., Epstein Jayaratne, Toby, and Anderson, Elizabeth S.. 2009. “Differentiating Contemporary Racial Prejudice from Old-Fashioned Racial Prejudice.” Race and Social Problems 1 (2): 97110.Google Scholar
Byrd, W. Carson, and Ray, Victor E.. 2015. “Ultimate Attribution in the Genetic Era White Support for Genetic Explanations of Racial Difference and Policies.” ANNALS of the American Academy of Political and Social Science 661 (1): 212–35.Google Scholar
Conrad, Peter. 2002. “The Double-Edged Helix: Social Implications of Genetics in a Diverse Society.” In Genetics and Behavior in the News: Dilemmas of a Rising Paradigm, eds. Asch, Adrienne, Beckwith, Jon, Conrad, Peter, Alper, Joseph S., Ard, Catherine, and Geller, Lisa N.. Baltimore, MD: Johns Hopkins Press, 5879.Google Scholar
Converse, Philip E. 1964. “The Nature of Belief Systems in Mass Publics”. In Ideology and Discontent, ed. Apter, David E.. Ann Arbor: University of Michigan Press pp. 206–61.Google Scholar
Dar-Nimrod, Ilan, and Heine, Steven J.. 2011. “Genetic Essentialism: On the Deceptive Determinism of DNA.” Psychological Bulletin 137 (5): 800–18.CrossRefGoogle ScholarPubMed
Dawson, Michael C. 2003. Black Visions: The Roots of Contemporary African-American Political Ideologies. Chicago: University of Chicago Press.Google Scholar
Dilliplane, Susanna, Goldman, Seth K., and Mutz, Diana C.. 2013. “Televised Exposure to Politics: New Measures for a Fragmented Media Environment.” American Journal of Political Science 57 (1): 236–48.CrossRefGoogle Scholar
Feldman, Stanley. 2013. “Political Ideology.” In The Oxford Handbook of Political Psychology, 2nd ed., vol. 2, eds. Sears, David O., Huddy, Leonie, and Levy, Jack S.. New York: Oxford University Press, 591626.Google Scholar
Garretson, Jeremiah, and Suhay, Elizabeth. 2016. “Scientific Communication about Biological Influences on Homosexuality and the Politics of Gay Rights.” Political Research Quarterly 69: 1729.Google Scholar
Gauchat, Gordon. 2012. “Politicization of Science in the Public Sphere: A Study of Public Trust in the United States, 1974 to 2010.” American Sociological Review 77 (2): 167–87.CrossRefGoogle Scholar
Goldman, Seth K., Mutz, Diana C., and Dilliplane, Susanna. 2013. “All Virtue is Relative: A Response to Prior.” Political Communication 30 (4): 635–53.Google Scholar
Haider-Markel, Donald P., and Joslyn, Mark R.. 2008. “Beliefs About the Origins of Homo-sexuality and Support For Gay Rights An Empirical Test of Attribution Theory.” Public Opinion Quarterly 72 (2): 291310.Google Scholar
Haider-Markel, Donald P., and Joslyn, Mark R.. 2014. “‘Nanny State’ Politics: Causal Attributions about Obesity and Support for Regulation.” American Politics Research. Online First.Google Scholar
Hart, P. Sol, and Nisbet, Erik C.. 2011. “Boomerang Effects in Science Communication: How Motivated Reasoning and Identity Cues Amplify Opinion Polarization About Climate Mitigation Policies.” Communication Research 39: 701–23.Google Scholar
Herrnstein, Richard J., and Murray, Charles. 1994. The Bell Curve: Intelligence and Class Structure in American Life. New York: Free Press.Google Scholar
Hochschild, Jennifer, and Sen, Maya. 2015a. “Genetic Determinism, Technology Optimism, and Race: Views of the American Public.” ANNALS of the American Academy of Political and Social Science 661 (September): 160–80.Google Scholar
Hochschild, Jennifer, and Sen, Maya. 2015b. “Technology Optimism or Pessimism about Genomic Science: Variation among Experts and Scholarly Disciplines.” ANNALS of the American Academy of Political and Social Science 658 (March): 236–52.Google Scholar
Hofstadter, Richard. 2006. Social Darwinism in American Thought. Boston: Beacon Press.Google Scholar
Hunt, Matthew O. 2007. “African American, Hispanic, and White Beliefs about Black/White Inequality, 1977–2004.” American Sociological Review 72 (3): 390415.Google Scholar
Iyengar, Shanto. 1991. Is Anyone Responsible? How Television Frames Political Issues. Chicago: University of Chicago Press.Google Scholar
Jacoby, Russell, and Glauberman, Naomi, eds. 1995. The Bell Curve Debate: History, Documents, Opinions. New York: Random House.Google Scholar
Jayaratne, Toby Epstein, Ybarra, Oscar, Sheldon, Jane P., Brown, Tony N., Feldbaum, Merle, Pfeffer, Carla A. and Petty, Elizabeth M.. 2006. “White Americans Genetic Lay Theories of Race Differences and Sexual Orientation: Their Relationship with Prejudice toward Blacks, and Gay Men and Lesbians.” Group Processes & Intergroup Relations 9 (1): 7794.Google Scholar
Jost, John T., Glaser, Jack, Kruglanski, Arie W., and Sulloway, Frank J.. 2003. “Political Conservatism as Motivated Social Cognition.” Psychological Bulletin 129 (3): 339.CrossRefGoogle ScholarPubMed
Kahan, Dan M. 2011. “Neutral Principles, Motivated Cognition, and Some Problems for Constitutional Law.” Harvard Law Review 125 (1): 178.Google Scholar
Kahan, Dan M. 2013. “Ideology, Motivated Reasoning, and Cognitive Reflection.” Judgment & Decision Making 8: 407–24.Google Scholar
Kahan, Dan M. 2015. “Climate-Science Communication and the Measurement Problem.” Advances in Political Psychology 36 (S1): 143.Google Scholar
Kinder, Donald R., and Sanders, Lynn M.. 1996. Divided by Color: Racial Politics and Democratic Ideals. Chicago: University of Chicago Press.Google Scholar
Kluegel, James R., and Smith, Eliot R.. 1986. Beliefs about Inequality: Americans’ Views of What Is and What Ought to Be. Hawthorne, NY: Aldine De Gruyter.Google Scholar
Kraft, Patrick W., Lodge, Milton, and Taber, Charles S.. 2015. “Why People Don't Trust the Evidence Motivated Reasoning and Scientific Beliefs.” ANNALS of the American Academy of Political and Social Science 658 (1): 121–33.Google Scholar
Kreuter, Frauke, Presser, Stanley, and Tourangeau, Roger. 2008. “Social Desirability Bias in CATI, IVR, and Web Surveys: The Effects of Mode and Question Sensitivity.” Public Opinion Quarterly 72 (5): 847–65.Google Scholar
Kunda, Ziva. 1990. “The Case for Motivated Reasoning.” Psychological Bulletin 108 (3): 480–98.Google Scholar
Lewontin, R. C., Rose, Steven, and Kamin, Leon J.. 1984. Not in Our Genes: Biology, Ideology, and Human Nature. New York: Pantheon Books.Google Scholar
Lodge, Milton, and Taber, Charles S.. 2013. The Rationalizing Voter. New York: Cambridge University Press.Google Scholar
Lord, Charles S., Ross, Lee, and Lepper, Mark. 1979. “Biased Assimilation and Attitude Polarization: The Effects of Prior Theories on Subsequently Considered Evidence.” Journal of Personality and Social Psychology 37: 2098–109.Google Scholar
Matthews, Dylan. 2016. “The alt-right is more than warmed-over white supremacy. It's that, but way way weirder.” Vox.com.Google Scholar
McKie, Robin. 2001. “Revealed: The Secret of Human Behaviour.” The Guardian, February 10.Google Scholar
Morgan, G. Scott, Mullen, Elizabeth, and Skitka, Linda J.. 2010. “When Values and Attributions Collide: Liberals and Conservatives Values Motivate Attributions for Alleged Misdeeds.” Personality and Social Psychology Bulletin 36: 1241–54.Google Scholar
Morin-Chassé, Alexandre. 2014. “Public (Mis)understanding of News about Behavioral Genetics Research: A Survey Experiment.” BioScience 64 (12): 1170–77.Google Scholar
Neville, Helen A., Lilly, Roderick L., Duran, Georgia, Lee, Richard M., and Browne, LaVonne. 2000. “Construction and Initial Validation of the Color-Blind Racial Attitudes Scale (CoBRAS).” Journal of Counseling Psychology 47 (1): 5970.Google Scholar
NHGRI (National Human Genome Research Institute). 2012. “Remarks Made by the President, Prime Minister Tony Blair of England (via satellite), Dr. Francis Collins, Director of the National Human Genome Research Institute, and Dr. Craig Venter, President and Chief Scientific Officer, Celera Genomics Corporation, on the Completion of the First Survey of the Entire Human Genome Project.” The White House, Office of the Press Secretary.Google Scholar
Nisbet, Erik C., Cooper, Kathryn E., and Garrett, R. Kelly. 2015. “The Partisan Brain: How Dissonant Science Messages Lead Conservatives and Liberals to (Dis)Trust Science.” ANNALS of the American Academy of Political and Social Science 658 (1): 3666.Google Scholar
Nisbet, Matthew C., and Scheufele, Dietram A.. 2009. “What's Next for Science Communication? Promising Directions and Lingering Distractions.” American Journal of Botany 96 (10): 1767–78.Google Scholar
Noel, Hans. 2013. Political Ideologies and Political Parties in America. New York: Cambridge University Press.Google Scholar
Pettigrew, Thomas F. 1979. “The Ultimate Attribution Error: Extending Allports Cognitive Analysis of Prejudice.” Personality and Social Psychology Bulletin 5 (4): 461–76.Google Scholar
Phelan, Jo C., Link, Bruce G., and Feldman, Naumi M.. 2013. “The Genomic Revolution and Beliefs about Essential Racial Differences A Backdoor to Eugenics?American Sociological Review 78: 167–91.Google Scholar
Ross, Lee. 1977. “Advances in Experimental Social Psychology.” In The Intuitive Psychologist and His Shortcomings: Distortions in the Attribution Process, vol. 10, ed. Berkowitz, Leonard. New York, NY: Academic Press, 173220.Google Scholar
Segerstrale, Ullica. 2000. Defenders of the Truth: The Battle for Science in the Sociobiology Debate and Beyond. New York: Oxford University Press.Google Scholar
Shen, Francis X., and Gromet, Dena M.. 2015. “Red States, Blue States, and Brain States Issue Framing, Partisanship, and the Future of Neurolaw in the United States.” ANNALS of the American Academy of Political and Social Science 658 (1): 86101.Google Scholar
Skitka, Linda J., Mullen, Elizabeth, Griffin, Thomas, Hutchinson, Susan, and Chamberlin, Brian. 2002. “Dispositions, Scripts, or Motivated Correction? Understanding Ideological Differences in Explanations for Social Problems.” Journal of Personality and Social Psychology 83 (2): 470–87.Google Scholar
Sniderman, Paul M., Brody, Richard A., and Tetlock, Philip E.. 1991. Reasoning and Choice: Explorations in Political Psychology. New York: Cambridge University Press.Google Scholar
Stroud, Natalie Jomini. 2011. Niche News: The Politics of News Choice. New York: Oxford University Press.Google Scholar
Suhay, Elizabeth, and Jayaratne, Toby Epstein. 2013. “Does Biology Justify Ideology? The Politics of Genetic Attribution.” Public Opinion Quarterly 77 (2): 497521.Google Scholar
Weiner, Bernard. 1995. Judgments of Responsibility: A Foundation for a Theory of Social Conduct. New York: Guilford Press.Google Scholar
Weiner, Bernard, Osborne, Danny, and Rudolph, Udo. 2011. “An Attributional Analysis of Reactions to Poverty: The Political Ideology of the Giver and the Perceived Morality of the Receiver.” Personality and Social Psychology Review 15: 199213.Google Scholar
Zaller, John R. 1992. The Nature and Origins of Mass Opinion. New York: Cambridge University Press.Google Scholar
Figure 0

Table 1. Genetic attributions regressed on ideology, media exposure, and their interaction

Figure 1

Figure 1. This figure presents the interaction between political ideology and self-reported recent exposure to news about genetics. Shaded areas represent 95% confidence intervals. Predicted values were derived from Model 2, Table 1.

Figure 2

Table 2. Effect of treatment & ideology on genetic attributions for black inequality

Figure 3

Figure 2. This figure presents the interaction between political ideology and exposure to the treatment news article. Shaded areas represent 95% confidence intervals. Predicted values were derived from Table 2, Model 2.

Figure 4

Table A1. Regression models by subgroups of media exposure (study 1)

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Table A2. Controlling for potential confounds of the political ideology term (study 1)

Figure 6

Table A3. Controlling for potential confounds of the media exposure term (study 1)

Figure 7

Table A4. Sample description (study 2)

Figure 8

Table A5. Regression models by experimental subgroups (study 2)

Figure 9

Table A6. Controlling for other interactions (study 2)

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Table A7. Ideology versus partisan identification (study 2)