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The Allergic Factor in Idiopathic Epilepsy

Published online by Cambridge University Press:  08 February 2018

D. C. Dewar
D. C. Dewar*
Affiliation:
Springfield Mental Hospital, London
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Spratling (1), writing in 1904, was the first to draw attention to the possible association of epilepsy with food sensitization. Previous to this observers had been impressed by the fall in fit incidence which occasionally accompanied intercurrent diseases, notably typhoid and tuberculosis. Hamilton (2), some years later, confirmed these observations in respect of intercurrent typhoid. It was not, however, till 1919 that Pagniez and Lieutaud (3) detailed the first authentic instances of epilepsy definitely ascribable to a specific food substance. These were two cases of sensitivity towards chocolate. The authors demonstrated that withdrawal of chocolate led to a cessation of seizures, and that these returned when it was re-introduced into the dietary.

Type
Research Article
Information
Journal of Mental Science , Volume 87 , Issue 369 , October 1941 , pp. 608 - 630
Copyright
Copyright © Royal College of Psychiatrists, 1941 

References

(1) Spratling, W. R. (1904), Epilepsy and its Treatment. W. B. Saunders Co., Philadelphia.CrossRefGoogle Scholar
(2) Hamilton, A. S. (1909), J.A.M.A., 53, 1902.Google Scholar
(3) Pagniez, and Lieutaud, (1919), La Presse Médicale, 27, 693.Google Scholar
(4) Wechsler, L. S. (1921), Med. R., 100, 714.Google Scholar
(5) Thomson, J. (1921), B.M.J., 2, 679.Google Scholar
(6) Ward, J. F. (1922), New York Med. J. and Med. Rec., 115, 593 Google Scholar
(7) Howell, L. P. (1923), Ohio State M. J., 19, 417.Google Scholar
(8) Rowe, A. H. (1927), J. Lab. Clin. Med., 13, 31.Google Scholar
(9) Spratling, R. H. (1931), J. Allergy, 3, 30.Google Scholar
(10) Forman, J. (1934), Arch. Neur. Psychiat., 32, 517.CrossRefGoogle Scholar
(11) Clein, N. W. (1937), North-west Med., 36, 378.Google Scholar
(12) Levin, S. J. (1931), J.A.M.A., 96, 1624.Google Scholar
(13) McReady, and Hay, (1924), M. J. and Rec. Suppl., 120, 117.Google Scholar
(14) Spangler, R. H. (1927), J. Lab. Clin. Med., 13, 41.Google Scholar
(15) Dewar, D. C. (1941), Journ. Ment. Sci., 87, 397.CrossRefGoogle Scholar
(16) Ward, and Patterson, (1927), Arch. Neurol. and Psychiat., 17, 427.Google Scholar
(17) Van Leeuwen, W. S. (1925), Allergic Diseases, p. 135. J. B. Lippincott & Co. Google Scholar
(18) Wallis, Nicol and Craig, (1923), Lancet, 1, 741.Google Scholar
(19) Singer, A. (1937), Wien. med. Wschr., 87, 1020.Google Scholar
(20) Crockett, (1921), B.M.J., 1, 458.Google Scholar
(21) Spangler, R. H. (1924), M. J. and Rec., 120, 495.Google Scholar
(22) Idem (1925). Arch. Int. Med., 36, 778.Google Scholar
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